A 45-year-old man with a severe traumatic brain injury is intubated and ventilated for rotor-wing transport to a level I trauma center. Shortly after lift-off and climb through 2,000 ft AGL, his pupillary response slows and his intracranial pressure monitor rises from 18 mm Hg to 30 mm Hg despite unchanged ventilator settings. Which factor related to the flight environment is the most likely cause of this acute increase in ICP?
Positive G-forces during climb impeding cerebral venous outflow
Hyperventilation failure leading to hypercarbia and cerebral vasodilation
Lower barometric pressure at altitude causing expansion of trapped intracranial gas
Rotor-blade vibration and cabin noise stimulating sympathetic discharge
As the helicopter ascends, ambient barometric pressure falls. According to Boyle's law, any intracranial air pocket (post-traumatic or post-operative pneumocephalus) expands as external pressure drops. The rigid skull cannot accommodate this volume increase, so ICP rises. Altitude-induced hypoxia further promotes cerebral vasodilation and edema, compounding the pressure elevation. Acceleration forces, sympathetic stimulation from noise/vibration, or equipment activation do not directly raise ICP to the same degree. Hypercarbia would increase ICP but was ruled out by stable ventilator settings and end-tidal COâ‚‚.
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BCEN CFRN
General Principles of Flight Transport Nursing Practice
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